Abstract While there has been considerable research on the patterns of pre- and post-substance use among individuals with traumatic brain injury (TBI), little is known about the incidence of co-morbid

The gap

Consensus across the literature

Supporting evidence — 3 representative gaps

• Sleep <scp>EEG</scp> as a Potential Marker of Alcoholism Predisposition—Commentary on “Adolescence and Parental History of Alcoholism: Insights from the Sleep <scp>EEG</scp>” (2012) · doi

  The extent to which these deficiencies contribute to future substance abuse remains to be determined, but the data highlight the need to take factors predating alcoholism onset, notably family history, into consideration before making causal assumptions between alcohol abuse and functional measures of brain activity.

  Keywords: abuse extent deficiencies contribute future substance remains determined highlight need take factors predating alcoholism onset
• The neurobiological and neurocognitive consequences of chronic cigarette smoking in alcohol use disorders (2007) · doi

  The material presented also contributes to ongoing discussions about treatment strategies for comorbid alcoholism and cigarette smoking and will hopefully stimulate further research into the neurobiological and neurocognitive consequences of chronic smoking in alcoholism and other substance use disorders.

  Keywords: alcoholism smoking material presented contributes ongoing discussions treatment strategies comorbid cigarette hopefully stimulate further neurobiological
• Co-Morbidity of Substance Abuse and Traumatic Brain Injury (2009) · doi

  Abstract While there has been considerable research on the patterns of pre- and post-substance use among individuals with traumatic brain injury (TBI), little is known about the incidence of co-morbidity between TBI and substance abuse (SA) in individuals whose primary disorder is SA.

  Keywords: substance individuals abstract there considerable patterns post among traumatic brain injury little known incidence morbidity