Biochemistry, Genetics and Molecular Biology · Research topic

Open research questions in Cellular transport and secretion

118 unresolved questions extracted from the limitations and future-work sections of 293 Cellular transport and secretion papers in our library. Each links back to the study that raised it.

What the literature leaves open

  • The lack of native storage-and-release architecture in non-specialized secretory cells. The limitation of classical secretion pathways in terms of response speed. The need for new design frontiers for synthetic secretion switches in mammalian cells.

    Beyond ER-Golgi trafficking: unconventional protein secretion as a new design frontier for synthetic secretion switches in mammalian cells · 2026 · DOI
  • dependence types26,54, cellular on In a sense, unconventional secretion provides a conceptually important but still underexplored direction for synthetic biology.

    Beyond ER-Golgi trafficking: unconventional protein secretion as a new design frontier for synthetic secretion switches in mammalian cells · 2026 · DOI
  • The mechanisms by which Spargel regulates endocycle-mediated growth have yet to be established. The relationship between Spargel and E2F1 in the regulation of cell growth is not well understood.

    Spargel/dPGC-1 influences cell growth through the E2F1-mediated endocycle pathway · 2026 · DOI
  • In order to flexibly change synaptic strength, the abundance of trans-synaptic adhesion complexes needs to be modulated, yet how this is achieved is poorly understood.

    The unhindered lateral mobility of a rhomboid intramembrane protease enables synapse remodelling · 2026 · DOI
  • Avl9 is involved in secretion and cell migration, but its molecular function has not been characterized.

    Avl9 defines a family of DENN domain GTPase-activating proteins · 2026 · DOI
  • Many GTPase regulators remain to be identified despite extensive genetic and biochemical screens.

    Avl9 defines a family of DENN domain GTPase-activating proteins · 2026 · DOI
  • While their importance is well-established, the precise dynamics of their conformational changes remain unclear.

    From Closed to Mighty: How GTP-Induced Allosteric Dynamics, Dimerization, and Membrane Binding Transform a Bacterial Dynamin-like Protein · 2026 · DOI
  • Moreover, transmitter loading of SVs accelerates fusion in vitro, which is abolished when synaptophysin is lacking despite near normal transmitter uptake.

    Synaptophysin accelerates synaptic vesicle fusion by expanding the membrane upon neurotransmitter loading · 2025 · DOI
  • The adaptor protein Sla1 inhibits Las17 but mechanisms underpinning Las17 activation remain elusive.

    Competitive binding of actin and SH3 domains at proline-rich regions of Las17/WASP regulates actin polymerisation · 2025 · DOI
  • However, the molecular basis for this TMED9-mediated cargo retention remains elusive.

    Molecular basis of TMED9 oligomerization and entrapment of misfolded protein cargo in the early secretory pathway · 2024 · DOI
  • However, the mechanism underlying fast SNARE-induced membrane fusion remains unknown.

    Molecular mechanism underlying SNARE-mediated membrane fusion enlightened by all-atom molecular dynamics simulations · 2024 · DOI
  • Until now, there is virtually no data on pyrophosphorylation of plant proteins, which is an exciting field that remains to be explored.

    Protein pyrophosphorylation by inositol phosphates: a novel post-translational modification in plants? · 2024 · DOI
  • While SCAMP genes have been studied in several plant species, their functions in cotton, particularly in response to abiotic stress, have not yet been reported.

    Genome‑wide analysis of cotton SCAMP genes and functional characterization of GhSCAMP2 and GhSCAMP4 in salt tolerance · 2024 · DOI
  • While the kinesin-3 family member KIF1C is established in mediating anterograde vesicle transport, its role in lysosomal transport remains unclear.

    KIF1C facilitates retrograde transport of lysosomes through Hook3 and dynein · 2024 · DOI
  • Despite its established function in cellular processes, its involvement in coronavirus replication remains unclear.

    Oxysterole-binding protein targeted by SARS-CoV-2 viral proteins regulates coronavirus replication · 2024 · DOI
  • Further exploration of the underlying mechanisms of Rab27 in tumors and exosomes. Investigation of the application value of Rab27 in antitumor therapeutic strategies. Development of novel small-molecule drugs targeting Rab27 for antitumor effects.

    Rab27: Molecular switch of tumor exosome secretion (Review) · 2026 · DOI
  • High turgor pressure in plant cells. Rigid cell walls in plant cells. Limited availability of specific inhibitors of plant CME.

    Clathrin‐Mediated Endocytosis in Plants: Historical to Modern Advances · 2026 · DOI
  • Many common inhibitors of CME have weak or no effects on plant CME. The mechanism of action of Ikarugamycin remains unknown. The study of plant CME is limited by the lack of specific inhibitors and the complexity of plant cellular constraints.

    Clathrin‐Mediated Endocytosis in Plants: Historical to Modern Advances · 2026 · DOI
  • To demonstrate enhanced interaction of TIO kinase with Kin-12A/B upon binding of RAB-A2a in vitro. To identify the molecular substrates of TIO kinase activity. To explain how TIO-dependent phosphorylation of its substrates promotes phragmoplast formation and expansion.

    A Rab/Kinesin-12/kinase module couples vesicle delivery and phragmoplast dynamics during plant cell cytokinesis · 2026 · DOI
  • Further investigation of the role of Rap1b in cAMP signaling. Exploration of the therapeutic potential of targeting Rap1b and AC9 in diseases related to cAMP signaling.

    Rap1 Activates Endosomal AC9 to Drive the Second cAMP Wave · 2026 · DOI
  • Further study of the role of PI3K complex II in endosomal clathrin removal and lysosomal maturation. Investigation of the effects of PI3K complex II depletion on other cellular processes. Development of therapies for diseases related to endosomal dysfunction.

    The Uvrag-containing PI3K complex promotes Hsc70-4 dependent endosomal clathrin removal and lysosomal maturation in Drosophila nephrocytes · 2026 · DOI
  • Further studies are needed to examine the long-term effects of Munc18.1 deficiency on cortical pyramidal cells. Studies should investigate the mechanisms underlying the stage-dependent effects of Munc18.1 deficiency. Research should examine the potential therapeutic strategies for neurodevelopmental disorders associated with Munc18.1 haploinsufficiency.

    Early and transient increase in cortical pyramidal cell excitability and delayed alteration of evoked synaptic transmission and t-SNARE proteins content in the hippocampus and neocortex of neonatal and juvenile Stxbp1 heterozygous mice · 2026 · DOI
  • The effects of Munc18.1 deficiency on intrinsic neuronal properties are not well understood. The developmental dependence of the effects of Munc18.1 deficiency is unknown.

    Early and transient increase in cortical pyramidal cell excitability and delayed alteration of evoked synaptic transmission and t-SNARE proteins content in the hippocampus and neocortex of neonatal and juvenile Stxbp1 heterozygous mice · 2026 · DOI
  • Further investigation of the mechanisms of PIKfyve regulation - Exploration of the potential therapeutic applications of enhancing PIKfyve activity - Examination of the role of PIKfyve in other cell types and diseases

    PIKfyve preserves endolysosomal function in photoreceptors and RPE cells to maintain retinal integrity · 2026 · DOI
  • The mechanisms by which photoreceptors eliminate misfolded or mistargeted proteins are not well understood - The specific role of disrupted PIP metabolism in retinal degenerative diseases remains poorly understood

    PIKfyve preserves endolysosomal function in photoreceptors and RPE cells to maintain retinal integrity · 2026 · DOI

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118 open questions have been extracted from the limitations and future-work passages of 293 Cellular transport and secretion papers in our library. Each one below links back to the study that raised it, so you can read the original claim in context.

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