Biochemistry, Genetics and Molecular Biology · Research topic

Open research questions in Diabetes and associated disorders

133 unresolved questions extracted from the limitations and future-work sections of 479 Diabetes and associated disorders papers in our library. Each links back to the study that raised it.

What the literature leaves open

  • The study did not account for potential confounding factors, - The study relied on genetic variants associated with GD and antibody-mediated immune-response phenotypes, - The study used a limited number of genetic variants as instrumental variables

    The causal relationship between antibody-mediated immune responses to infections and Graves’ disease: a bi-directional Mendelian randomization analysis · 2026 · DOI
  • Further investigation into the relationship between antibody-mediated immune responses and GD risk, - Examination of the potential influence of antibody-mediated immune responses on GD risk, - Investigation into the mechanisms underlying the association between GD and anti-polyomavirus 2 IgG seropositivity

    The causal relationship between antibody-mediated immune responses to infections and Graves’ disease: a bi-directional Mendelian randomization analysis · 2026 · DOI
  • further study of the role of Lyn-deficiency in disease initiation, - investigation of the effects of environmental factors on disease development, - comparison of the model to other mouse models of uveoretinitis

    Spontaneous organ-specific eye autoimmune disease resulting from two engineered gene mutations compromising immune tolerance · 2026 · DOI
  • The gap in understanding how genetic compromises in multiple immune tolerance mechanisms confer susceptibility to autoimmune disease development. The need for a better understanding of the mechanisms of disease initiation. The lack of a comprehensive model of spontaneously developing organ-specific autoimmune disease.

    Spontaneous organ-specific eye autoimmune disease resulting from two engineered gene mutations compromising immune tolerance · 2026 · DOI
  • Compared to HLA-A and HLA-B, relatively little is known about the participation of HLA-C-restricted T cells in T1D.

    Islet-derived T cells from both mice and humans recognize conserved insulin A-chain peptides presented by HLA-C*03:04 · 2026 · DOI
  • This reinforces the link between emphysema, pulmonary TLT formation, and B cells; however, the exact mechanism involving B cells and pulmonary TLT in the development of emphysema remains elusive.

    A mutation in PTPN6 associated to emphysema alters B-lymphocyte biology in humans and mice · 2026 · DOI
  • Cell type–specific regulatory programs that drive type 1 diabetes (T1D) in the pancreas are poorly understood.

    Single-cell multiome and spatial profiling reveals pancreas cell type–specific gene regulatory programs of type 1 diabetes progression · 2025 · DOI
  • Furthermore, the relationship between infection severity and new autoimmune disease risk has not been well examined.

    Severity of acute SARS-CoV-2 infection and risk of new-onset autoimmune disease: A RECOVER initiative study in nationwide U.S. cohorts · 2025 · DOI
  • These findings indicate that HIF-1α plays a diminished role in controlling IL-10 production in NOD B cells and that IL-10 alone is insufficient to maintain immune tolerance.

    Altered B cell metabolic pathways characterize type 1 diabetes progression · 2025 · DOI
  • Defects in Breg development and function have been identified in both human T1D and the non-obese diabetic (NOD) mouse, but whether these impairments reflect intrinsic B-cell abnormalities or microenvironmental changes associated with hyperglycemia and inflammation remains unclear.

    Altered B cell metabolic pathways characterize type 1 diabetes progression · 2025 · DOI
  • Advances in restriction fragment length polymorphism (RFLP) analysis in the mid-1980s provided evidence that HLA-DQ genes were more strongly associated with T1D than HLA-DR,13–19 but direct sequencing data were lacking.

    Decoding autoimmunity: HLA-DQβ and type 1 diabetes · 2025 · DOI
  • T1D incidence is increasing worldwide but it is poorly understood what activates autoreactive T-cells.

    The impact of Parabacteroides distasonis colonization on the microbiome, metabolome, immune responses in SPF mice and diabetes onset in germ-free mice 4785 · 2025 · DOI
  • However, how Ptpn22 impacts B cell dynamics during virus infection remains poorly understood.

    PTPN22 impacts the B cell landscape during chronic virus infection 4379 · 2025 · DOI
  • While the exact cause remains unknown, studies in mouse models indicate that sympathetic denervation and macrophage depletion can halt T1D progression, suggesting a critical role for neuroimmune communication in the disease’s development.

    Neuroimmune interactions in the pancreas in health and disease- Identifying the components of nerve-macrophage association 2911 · 2025 · DOI
  • While preventive measures to manage periodontal complications in T1DM patients may improve overall health outcomes, further research is needed to understand the intricate interactions between oral microbiota, host response, periodontal disease, and systemic health in this population.

    Investigating the Interplay: Periodontal Disease and Type 1 Diabetes Mellitus—A Comprehensive Review of Clinical Studies · 2024 · DOI
  • However, the precise trigger of autoimmunity and disease onset remain incompletely elucidated.

    Gene-environment interaction in the pathophysiology of type 1 diabetes · 2024 · DOI
  • Extracellular vesicles (EVs) from β cells have been shown to carry protein and microRNAs (miRNAs), influencing cellular signaling and may contribute to the development of T1D but it remains to be addressed how senescence impacts β cell EV cargo.

    Exploring senescence as a modifier of β cell extracellular vesicles in type 1 diabetes · 2024 · DOI
  • Objective: Studies have shown an increased incidence of pediatric type 1 diabetes during the COVID-19 pandemic, but the detailed role of SARS-CoV-2 infection in the incidence increase in type 1 diabetes remains unclear.

    Spatiotemporal association between COVID-19 incidence and type 1 diabetes incidence among children and adolescents: a register-based ecological study in Germany · 2024 · DOI
  • However, further studies with large sample sizes and validation across different racial and ethnic populations are warranted.

    Case report: Strong GAD antibody positivity and type 1 diabetes-HLA-susceptible haplotype-DRB1*04:05-DQB1*04:01 in a Japanese patient with immune checkpoint inhibitor-induced type 1 diabetes · 2024 · DOI
  • Background The functional changes in alpha cells in patients with type 1 diabetes (T1D) with different residual beta cell functions remain poorly elucidated.

    Abnormal late postprandial glucagon response in type 1 diabetes is a function of differences in stimulated C-peptide concentrations · 2024 · DOI
  • Although several studies have indicated that fatty acid mitochondrial oxidation is critical for immune cell functions, contradictory findings have been reported.

    CPT2-mediated Fatty Acid Oxidation Is Dispensable for Humoral Immunity · 2024 · DOI
  • Although several neutralizing Abs against a small number of HPIV3 F epitopes have been identified to date, relatively little is known about the Ab response to HPIV3 compared with other pathogens, such as influenza virus and SARS-CoV-2.

    Potent HPIV3-neutralizing IGHV5-51 Antibodies Identified from Multiple Individuals Show L Chain and CDRH3 Promiscuity · 2024 · DOI
  • There is a need to determine the time to resolution of diabetic ketoacidosis and its predictors among adult diabetic patients in public hospitals of Northeast Ethiopia. There is a need to identify predictors of time to resolution of diabetic ketoacidosis.

    Time to Resolution of Diabetic Ketoacidosis and Its Predictors among Adult Diabetic Patients in Public Hospitals of Northeast, Ethiopia · 2026 · DOI
  • ARTICLE IN PRESS ARTICLE IN PRESS ACCEPTED MANUSCRIPT This study has several limitations. Using secondary data, we were unable to assess certain predictors of time to DKA resolution, including admission pH, potassium levels, and adherence to treatment protocols. The analysis focused on the number of DKA episodes rather than individual patients, so those with multiple episodes may have been included more than once, potentially affecting results due to varying clinical characteristics and treatment responses. Data on baseline or chronic medications, including SGLT-2 inhibitors, were unavailable, limiting adjustment for potential confounders. Furthermore, missing physiological data—such as pH, bicarbonate, anion gap, creatinine, and lactate prevented adjustment for these variables, despite guidelines recommending pH >7.3, bicarbonate ≥18 mmol/L, and anion gap closure as standard criteria for DKA resolution.

    Time to Resolution of Diabetic Ketoacidosis and Its Predictors among Adult Diabetic Patients in Public Hospitals of Northeast, Ethiopia · 2026 · DOI
  • Limited evidence from randomised trials in transplant recipients. Variability in trial duration and enrolment criteria. Incompletely and variably reported outcomes.

    Comparison of incidence patterns and risk profiles of ST-elevation and non-ST-elevation myocardial infarction in type 1 diabetes in Finland: a nationwide cohort study · 2026 · DOI

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133 open questions have been extracted from the limitations and future-work passages of 479 Diabetes and associated disorders papers in our library. Each one below links back to the study that raised it, so you can read the original claim in context.

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